Search results for " beta 3"

showing 4 items of 4 documents

Intracellular accumulation of incompletely processed transforming growth factor-alpha polypeptides in ground glass hepatocytes of chronic hepatitis B…

1996

Abstract Background: Transforming growth factor-alpha is an intracellularly processed and secreted polypeptide that induces a proliferative response in epithelial target cells and represents a potential regulatory factor in embryonic development, liver regeneration, and also hepatocarcinogenesis. We have observed focal transforming growth factor-alpha expression in liver tissues with chronic hepatitis B virus infection. Methods: To further elucidate the nature of this focal transforming growth factor-alpha accumulation were have analyzed overall 23 different liver tissues with chronic hepatitis B virus and hepatitis C virus infection as well as normal liver tissues by immunohistology, ELISA…

TGF alphaHepatitis B Surface AntigensHepatologybiologyHepatitis C virusGrowth factormedicine.medical_treatmentHepatitis BTransforming Growth Factor alphamedicine.disease_causemedicine.diseasebiology.organism_classificationHepatitis BVirologyMolecular biologyLiver regenerationHepadnaviridaeLiverTransforming growth factor beta 3Case-Control StudiesChronic DiseasemedicineHumansGDF15PeptidesJournal of hepatology
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TGF-β superfamily signaling is essential for tooth and hair morphogenesis and differentiation

2007

Members of the transforming growth factor beta (TGF-beta) superfamily of signaling molecules are involved in the regulation of many developmental processes that involve the interaction between mesenchymal and epithelial tissues. Smad7 is a potent inhibitor of many members of the TGF-beta family, notably TGF-beta and activin. In this study, we show that embryonic overexpression of Smad7 in stratified epithelia using a keratin 5 promoter, results in severe morphogenetic defects in skin and teeth and leads to embryonic and perinatal lethality. To further analyze the functions of Smad7 in epithelial tissues of adult mice, we used an expression system that allowed a controlled overexpression of …

Cell signalingmedicine.medical_specialtyHistologyMorphogenesisEmbryonic DevelopmentMice TransgenicNerve Tissue ProteinsBiologySmad7 ProteinPathology and Forensic MedicineNestinMice03 medical and health sciences0302 clinical medicineIntermediate Filament ProteinsGenes ReporterTransforming Growth Factor betaInternal medicineMorphogenesismedicineAnimalsHumansTransgenes030304 developmental biology0303 health sciencesR-SMADIntegrasesintegumentary systemTooth Abnormalities[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyCell DifferentiationCell BiologyGeneral MedicineHair follicleSurvival AnalysisCell biologyKeratin 5Endocrinologymedicine.anatomical_structureGene Expression RegulationLac OperonTransforming growth factor beta 3030220 oncology & carcinogenesisRabbitsAmeloblastToothHairSignal TransductionTransforming growth factorEuropean Journal of Cell Biology
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Contribution of sinusoidal endothelial liver cells to liver fibrosis: expression of transforming growth factor-beta 1 receptors and modulation of pla…

1993

Transforming growth factor-beta 1 is an important cytokine in the pathophysiology of liver fibrosis, stimulating the production of extracellular matrix. Whether this cytokine can also control the degradation of matrix proteins in liver cells has not been investigated. Because plasmin is an important protease for the degradation of matrix glycoproteins, we investigated whether sinusoidal endothelial liver cells could contribute to fibrosing liver disease through the modulation of plasmin-generating enzymes in response to transforming growth factor-beta 1. Sinusoidal endothelial cells from guinea pig liver were investigated in pure monolayer culture. Using 125I-labelled transforming growth fa…

PlasminGuinea PigsBiologyLiver Cirrhosis Experimental03 medical and health sciencesPlasminogen Activators0302 clinical medicineCell surface receptorTransforming Growth Factor betaPlasminogen Activator Inhibitor 1medicineAnimalsFibrinolysinCells Cultured030304 developmental biology0303 health sciencesHepatology3. Good healthCell biologyFibronectinEndothelial stem cellBiochemistryLiverTransforming growth factor beta 3Cell culturebiology.protein030211 gastroenterology & hepatologyFemaleEndothelium VascularPlasminogen activatorReceptors Transforming Growth Factor betamedicine.drugTransforming growth factorHepatology (Baltimore, Md.)
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Evidence for transforming growth factor-beta 3 gene polymorphism in non-syndromic cleft lip and palate patients from indian sub-continent

2011

Objectives: Orofacial clefts are major human birth defects with complex etiology. Previous studies have proposed Transforming growth factor - beta 3 (TGF-β3) gene as a key player in contributing to non-syndromic cleft lip and palate, however none of the studies have yet included Indian population. Hence this study was designed to detect TGF-β3 gene polymorphism in nonsyndromic cleft lip and palate patients from Indian population which is genetically distinct from previously studied populations. Study Design: Peripheral blood samples of forty non-syndromic cleft lip and palate patients and forty unaffected individuals were collected for a case – control study design. Ethical clearance from t…

Beta-3 adrenergic receptorCleft LipDentistryIndiaBioinformaticslaw.inventionTransforming Growth Factor beta3lawMedicineHumansGeneral DentistryGenePolymerase chain reactionOral Medicine and PathologyPolymorphism Geneticbusiness.industryInstitutional review board:CIENCIAS MÉDICAS [UNESCO]Cleft PalateOtorhinolaryngologyTransforming growth factor beta 3UNESCO::CIENCIAS MÉDICASEtiologySurgeryResearch-ArticleGene polymorphismbusinessNon syndromic
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